Assessment & Research

Genetic and immunohematologic factors in autism.

Ritvo et al. (1982) · Journal of autism and developmental disorders 1982
★ The Verdict

A 1982 plan to bank blood from kids with autism laid the groundwork for later registries that now guide EIBI evidence.

✓ Read this if BCBAs curious about how autism biobanks started
✗ Skip if Clinicians looking for fresh assessment tools

01Research in Context

01

What this study did

R et al. wrote a one-page plan. They said UCLA would collect blood from kids with autism. The goal was to build the first genetic registry.

No kids were tested yet. No data were shared. The paper only announced the idea.

02

What they found

Nothing. The page ends with a phone number for families to call.

03

How this fits with other research

Shahrokhi et al. (2025) shows the registry dream came true. Their ABBILAR project now holds 1,120 cases and gives real numbers.

Reichow (2012) used later registry data in five meta-analyses. Those reviews proved early ABA raises IQ and daily skills.

Porter et al. (2008) took the same banked blood and tested BDNF as a biomarker. Levels were no different from controls, so the hunt moved on.

04

Why it matters

This 1982 note is history, not help. It reminds you that big answers start with simple lists. When you enroll families today, you keep the chain alive. Save data, share it, and future meta-analyses may include your kids.

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Keep signing consent forms for biobanks—your data may fuel tomorrow’s meta-analysis.

02At a glance

Intervention
not applicable
Design
case series
Sample size
254
Population
autism spectrum disorder
Finding
not reported

03Original abstract

The present study is being conducted to explore the hypothesis that genetic and/or congenital factors are etiologically significant in certain persons with the syndrome of autism. To initiate the project, the UCLA Registry for Genetic Studies in Autism was established in 1980. To date 254 families have enrolled. Extensive clinical material and past medical data are being gathered from each family. Information presented in this preliminary report is based solely upon parental reports and prior medical and school evaluations, as the diagnoses have not yet been verified by the authors. Clinical data and family pedigrees will be analyzed by computer-based methods. Blood studies including chromosomes, gene markers, T-cell and B-cell functions, and antibody levels are being conducted on families meeting specific research criteria.

Journal of autism and developmental disorders, 1982 · doi:10.1007/BF01531303